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當(dāng)前位置:華瑞科研網(wǎng)-ELISA試劑盒>>科研抗體>>科研一抗>>Anti-C1Q抗體,補(bǔ)體C1qα鏈多肽抗體科研

Anti-C1Q抗體,補(bǔ)體C1qα鏈多肽抗體科研

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更新時(shí)間:2018-05-18 15:37:56瀏覽次數(shù):290次

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商鋪產(chǎn)品:9992條

所在地區(qū):上海上海市

聯(lián)系人:韓經(jīng)理 (銷售)

產(chǎn)品簡(jiǎn)介

“Anti-C1Q抗體,補(bǔ)體C1qα鏈多肽抗體科研“我公司擁有*的細(xì)胞服務(wù)技術(shù),*的儀器設(shè)備和專業(yè)的ELISA檢測(cè)試劑盒、生物索標(biāo)記、熒光索標(biāo)記、酶標(biāo)記等,廣泛用于多種分析研究與技術(shù)測(cè)定。咨詢,我們將竭誠(chéng)為你服務(wù)!

詳細(xì)介紹

Anti-C1Q抗體,補(bǔ)體C1qα鏈多肽抗體科研產(chǎn)品介紹:

C1q, a subcomponent of the classical complement pathway, is composed of nine subunits that mediate classical complement activation and thereby play an important role in the immune response. Six of these subunits are disulfide-linked dimers of chains A and B, while three of these subunits, designated C1q-A through C1q-C, are disulfide-linked dimers of chain C. The presence of receptors for C1q on effector cells modulates its activity, which may be antibody-dependent or independent. Macrophages are the primary source of C1q, while anti-inflammatory drugs as well as cytokines differentially regulate expression of the mRNA as well as the protein. However, its ability to modulate the interaction of plaets with collagen and immune complexes suggests C1q influences homeostasis as well as other immune activities, and perhaps thrombotic complications resulting from immune injury. Defects in C1q-A, C1q-B and C1q-C cause inactivation of the classical pathway, leading to a rare genetic disorder characterized by lupus-like symptoms. 


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產(chǎn)品詳情:
產(chǎn)品編號(hào):HRK-11336R
產(chǎn)品規(guī)格: 0.1ml/0.2ml
產(chǎn)品別名:C1QA; C1QA_HUMAN; C1QB; C1QC; Complement C1q subcomponent subunit A; Complement C1q subcomponent subunit B; Complement C1q subcomponent subunit C; Complement component 1 q subcomponent A chain; Complement component 1 q subcomponent alpha polypeptide; Complement component C1q A chain; Complement component C1q B chain; Complement component C1q C chain
產(chǎn)品價(jià)格:詢價(jià)(電詢或客服)
產(chǎn)品用途:科研實(shí)驗(yàn)
貯 存:   貯存于-20℃.

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